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    <dc:creator>Michael.Massing</dc:creator><dc:subject>diet dementia foods risk anti-inflammatory pro-inflammatory correlation human in vivo clinical trial</dc:subject>
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    <title>Is Tofu Good for You? - The New York Times</title>
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    <dc:creator>Michael.Massing</dc:creator><description><![CDATA[Dr. Sun...led a study published in 2020 that found that consuming more soy isoflavones, particularly from tofu, was associated with a moderately lower risk of coronary heart disease.

And in another study, of nearly 120,000 health care professionals in the United States, Dr. Sun and his colleagues found that during the more than 30 years of follow-up, those who consumed at least one serving of tofu or soy milk per week were 15 to 16 percent less likely to die than those who ate less than one serving per month.]]></description>
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<item rdf:about="https://www.wsws.org/en/articles/2023/10/10/covi-o10.html">
    <title>Study finds that SARS-CoV-2 can infect the arteries of the heart - World Socialist Web Site</title>
    <dc:date>2023-10-12T19:23:22+00:00</dc:date>
    <link>https://www.wsws.org/en/articles/2023/10/10/covi-o10.html</link>
    <dc:creator>Michael.Massing</dc:creator><dc:subject>clinical cohort COVID19 human in mortality heart cardiovascular arteries plaque research retrospective risk treatment vivo</dc:subject>
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<dc:identifier>https://pinboard.in/u:Michael.Massing/b:eda87666376a/</dc:identifier>
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</item>
<item rdf:about="https://www.thelancet.com/journals/lanwpc/article/PIIS2666-6065(23)00235-3/fulltext#%20">
    <title>Effectiveness of community-based oral antiviral treatments against severe COVID-19 outcomes in people 70 years and over in Victoria, Australia, 2022: an observational study - The Lancet Regional Health – Western Pacific</title>
    <dc:date>2023-10-12T18:48:13+00:00</dc:date>
    <link>https://www.thelancet.com/journals/lanwpc/article/PIIS2666-6065(23)00235-3/fulltext#%20</link>
    <dc:creator>Michael.Massing</dc:creator><dc:subject>COVID19 mortality risk treatment Paxlovid clinical research in vivo human retrospective cohort</dc:subject>
<dc:source>https://pinboard.in/</dc:source>
<dc:identifier>https://pinboard.in/u:Michael.Massing/b:75dcd7afbe3b/</dc:identifier>
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<item rdf:about="https://www.nytimes.com/article/air-quality-index.html?name=styln-wildfires-smoke&amp;region=TOP_BANNER&amp;block=storyline_menu_recirc&amp;action=click&amp;pgtype=LegacyCollection&amp;variant=undefined">
    <title>What is the Air Quality Index? Here's How to Stay Safe - The New York Times</title>
    <dc:date>2023-06-07T05:27:02+00:00</dc:date>
    <link>https://www.nytimes.com/article/air-quality-index.html?name=styln-wildfires-smoke&amp;region=TOP_BANNER&amp;block=storyline_menu_recirc&amp;action=click&amp;pgtype=LegacyCollection&amp;variant=undefined</link>
    <dc:creator>Michael.Massing</dc:creator><dc:subject>air pollution quality index AQI risk mitigation</dc:subject>
<dc:source>https://pinboard.in/</dc:source>
<dc:identifier>https://pinboard.in/u:Michael.Massing/b:68180dd61f99/</dc:identifier>
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</item>
<item rdf:about="https://www.washingtonpost.com/news/wonk/wp/2015/05/14/the-safest-and-deadliest-ways-to-travel/">
    <title>The safest — and deadliest — ways to travel - The Washington Post</title>
    <dc:date>2023-06-03T01:08:49+00:00</dc:date>
    <link>https://www.washingtonpost.com/news/wonk/wp/2015/05/14/the-safest-and-deadliest-ways-to-travel/</link>
    <dc:creator>Michael.Massing</dc:creator><dc:subject>long-distance travel relative risk fatality death mode transportation statistics</dc:subject>
<dc:source>https://pinboard.in/</dc:source>
<dc:identifier>https://pinboard.in/u:Michael.Massing/b:a5a4afca7e83/</dc:identifier>
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</item>
<item rdf:about="https://turbli.com/blog/the-safest-transport-modes-ranked-by-statistics-from-10-years-of-data/">
    <title>The Safest Transport Modes, Ranked by Statistics From 10 Years of Data - turbli</title>
    <dc:date>2023-06-03T01:05:23+00:00</dc:date>
    <link>https://turbli.com/blog/the-safest-transport-modes-ranked-by-statistics-from-10-years-of-data/</link>
    <dc:creator>Michael.Massing</dc:creator><dc:subject>long-distance travel relative risk fatality death mode transportation statistics</dc:subject>
<dc:source>https://pinboard.in/</dc:source>
<dc:identifier>https://pinboard.in/u:Michael.Massing/b:be0d18cdbe17/</dc:identifier>
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</item>
<item rdf:about="https://www.wired.com/story/aging-brain-volume/">
    <title>The Modern World Is Aging Your Brain | WIRED</title>
    <dc:date>2023-05-22T03:57:20+00:00</dc:date>
    <link>https://www.wired.com/story/aging-brain-volume/</link>
    <dc:creator>Michael.Massing</dc:creator><description><![CDATA[Four years and 1,165 participants later, the results revealed a stark difference. When compared to similar data taken from the US and Europe, the Tsimane fare much better, especially in old age. Tsimane brains lose about 2.3 percent of their volume per decade, compared with around 2.8 percent for the Moseten and about 3.5 percent for industrialized populations. For septuagenarians and older, the difference nearly doubled.

In industrialized populations, brain volume usually drops with increasing body mass index and non-HDL (so-called “bad”) cholesterol. But Tsimane and Moseten brain volumes largely increased with rising BMI and cholesterol. Kaplan believes this discrepancy makes sense given humankind’s evolutionary past. If you have to work a lot to get your food, more energy actually helps. The Tsimane walk 17,000 steps per day. Older Tsimane provide food and care for their grandchildren and don’t really retire, Kaplan says. People who live in the US and Europe on average work much less for their calories, which creates a surplus.

To Kaplan, the new data suggests a “sweet spot” between energy in and energy out and that it’s fine to build up a higher BMI if you expend a lot of energy too. But without that balance, you may lose brain volume faster, perhaps due to poorer cardiovascular health—although the exact mechanism remains unclear. “We’re at the point where we’ve overshot the mark,” he says of industrialized populations. “We have too many calories, too little physical activity, leading to negative effects on our brains.” The team refers to the phenomenon as the “embarrassment of riches” hypothesis.]]></description>
<dc:subject>brain cardiovascular health risk benefit lifestyle culture BMI cholesterol blood lipids comparative physiology metabolism</dc:subject>
<dc:source>https://pinboard.in/</dc:source>
<dc:identifier>https://pinboard.in/u:Michael.Massing/b:e22e31e80619/</dc:identifier>
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</item>
<item rdf:about="https://www.google.com/search?client=safari&amp;rls=en&amp;q=%22Prediabetic%22+was+a+negotiated+label+for+failing+a+one+of+two+oral+glucose+tolerance+tests#ip=1">
    <title>&quot;Prediabetic&quot; was a negotiated label for failing a one of two oral glucose tolerance tests - Google Search</title>
    <dc:date>2023-04-29T18:28:09+00:00</dc:date>
    <link>https://www.google.com/search?client=safari&amp;rls=en&amp;q=%22Prediabetic%22+was+a+negotiated+label+for+failing+a+one+of+two+oral+glucose+tolerance+tests#ip=1</link>
    <dc:creator>Michael.Massing</dc:creator><dc:subject>diabetes type 2 T2D diagnosis threshold morbidity mortality comorbidity risk overview criticism standard</dc:subject>
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<dc:identifier>https://pinboard.in/u:Michael.Massing/b:7e811b2f162f/</dc:identifier>
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<item rdf:about="https://medicalxpress.com/news/2023-04-diabetes.html">
    <title>New test could help identify type 2 diabetes risk</title>
    <dc:date>2023-04-07T22:15:39+00:00</dc:date>
    <link>https://medicalxpress.com/news/2023-04-diabetes.html</link>
    <dc:creator>Michael.Massing</dc:creator><dc:subject>diabetes type 2 research peer-reviewed human in vivo clinical situ screening prevention delay risk factor</dc:subject>
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</item>
<item rdf:about="https://www.sflorg.com/2023/03/bio03282302.html">
    <title>Scientific Frontline: Adipose tissue as a culprit: How obesity leads to diabetes</title>
    <dc:date>2023-03-28T21:50:07+00:00</dc:date>
    <link>https://www.sflorg.com/2023/03/bio03282302.html</link>
    <dc:creator>Michael.Massing</dc:creator><dc:subject>type 2 diabetes T2D insulin resistance fat dietary adipose tissue pathophysiology risk factor etiology diet behavior behavioral environment environmental obesity endocrine body</dc:subject>
<dc:source>https://pinboard.in/</dc:source>
<dc:identifier>https://pinboard.in/u:Michael.Massing/b:77e14458fb43/</dc:identifier>
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<item rdf:about="https://www.levelshealth.com/blog/do-you-get-enough-zinc-heres-how-it-benefits-metabolic-health">
    <title>Do you get enough zinc? Here’s how it benefits metabolic health - Levels</title>
    <dc:date>2023-03-24T15:02:04+00:00</dc:date>
    <link>https://www.levelshealth.com/blog/do-you-get-enough-zinc-heres-how-it-benefits-metabolic-health</link>
    <dc:creator>Michael.Massing</dc:creator><description><![CDATA[According to the National Institutes of Health, the daily zinc requirements are:
Men, age 19+: 11 milligrams (mg)
Women, age 19+: 8 mg (11 mg if pregnant)
You can supplement with more zinc than the RDA since the RDA represents the minimum required amount of zinc, which may be lower than optimal zinc levels, mainly due to large-scale factors that put the general population at higher risk for zinc deficiency. Mark Hyman, MD, a Levels Advisor and best-selling author, recommends taking 30 mg daily for men and women.
The general guidance for the upper limit for zinc intake is 40 mg daily since excessive zinc intake can cause symptoms such as nausea, dizziness, headaches, and loss of appetite and is associated with high blood pressure, reduced immune function, and lower HDL cholesterol levels. Studies have also shown that very high doses of zinc from supplements (142 mg/day) may interfere with magnesium and copper absorption.]]></description>
<dc:subject>T2D pancreas beta cells wound healing zinc dosage overview risk benefit systemic effects diabetes type 2 interactions food sources blood levels</dc:subject>
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</item>
<item rdf:about="https://www.rush.edu/news/understanding-link-between-covid-19-and-diabetes">
    <title>Understanding the Link Between COVID-19 and Diabetes | Rush System</title>
    <dc:date>2023-01-10T17:15:30+00:00</dc:date>
    <link>https://www.rush.edu/news/understanding-link-between-covid-19-and-diabetes</link>
    <dc:creator>Michael.Massing</dc:creator><description><![CDATA[Scientists already know that for some people, it’s not just a faulty immune system that triggers type 1 diabetes or insulin resistance that leads to type 2 diabetes. The National Institute of Diabetes, Digestive and Kidney Diseases recognizes more than 50 different ways that a person can develop diabetes, from defects in a single gene to other health issues like liver disease.

“Ideally, we would like to investigate each and every one of those 50 causes,” Kazlauskaite says. “And we are finding that some people have several diabetes causes at the same time. In other words, a patient may not just have the purely insulin-resistant type of diabetes, what’s typically defined as type 2, but they might also be affected by steroids or autoimmune diabetes at the same time.” Doctors may call this “hybrid diabetes,” suggesting that the etiology, or cause, of diabetes may be much more complex than originally believed.

Could COVID-19 be a new cause of diabetes?
It’s possible, Kazlauskaite says. A study recently published in the medical journal Lancet found that people who survived COVID have a 40% higher risk for developing diabetes. She believes many of the new cases of type 2 diabetes are occurring in people with pre-diabetes who don’t know it.

“People might have pre-diabetes with chronic, subclinical inflammation before they get COVID-19. So, when they do get infected with the virus, they could develop hyperinflammation that causes metabolic stress and more insulin resistance. As a result, they manifest overt diabetes,” she says.

Another theory is that COVID-19 disrupts the immune system. “We know hyperinflammation can cause autoimmune disease. When this happens, the immune system gets ‘confused’ and starts attacking its own tissues, including insulin-producing cells in the pancreas,” she says. This is the process that causes type 1 diabetes, which primarily develops in childhood but may also affect adults.

A third theory is that steroids given during COVID-19 treatment could cause diabetes because they often raise blood glucose, Kazlauskaite says. For some patients, glucose levels return to normal after the steroids have been discontinued, but for others, the effects are lasting.

A fourth reported hypothesis is that COVID-19 can cause severe inflammation of the pancreas (known as pancreatitis), where the body’s insulin-producing cells reside. This can trigger diabetes, although it is relatively rare]]></description>
<dc:subject>diabetes etiology cause risk factors type 1 2 T1D T2D autoimmune hyperinflammation prediabetes inflammation subclinical overview resarch long COVID post-acute sequelae liver hybrid insulin resistance immune system steroids pancreatitis pancreas metabolic stress</dc:subject>
<dc:source>https://pinboard.in/</dc:source>
<dc:identifier>https://pinboard.in/u:Michael.Massing/b:6fbe3726f6ad/</dc:identifier>
<taxo:topics><rdf:Bag>	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:diabetes"/>
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	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:T1D"/>
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	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:liver"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:hybrid"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:insulin"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:resistance"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:immune"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:system"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:steroids"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:pancreatitis"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:pancreas"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:metabolic"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:stress"/>
</rdf:Bag></taxo:topics>
</item>
<item rdf:about="https://www.medrxiv.org/content/10.1101/2022.12.21.22283753v1">
    <title>Outpatient treatment of Covid-19 with metformin, ivermectin, and fluvoxamine and the development of Long Covid over 10-month follow-up. | medRxiv</title>
    <dc:date>2022-12-26T01:59:34+00:00</dc:date>
    <link>https://www.medrxiv.org/content/10.1101/2022.12.21.22283753v1</link>
    <dc:creator>Michael.Massing</dc:creator><description><![CDATA[There was a 42% relative decrease in the incidence of Long Covid in the metformin group compared to its blinded control in a secondary outcome of this randomized phase 3 trial.]]></description>
<dc:subject>metformin preprint research clinical trial risk comparison human in vivo randomized phase 3</dc:subject>
<dc:source>https://pinboard.in/</dc:source>
<dc:identifier>https://pinboard.in/u:Michael.Massing/b:4c72b7923adf/</dc:identifier>
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	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:randomized"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:phase"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:3"/>
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</item>
<item rdf:about="https://www.diabetesselfmanagement.com/news-research/2022/12/14/a1c-testing-may-help-detect-undiagnosed-diabetes/">
    <title>A1C Testing May Help Detect Undiagnosed Diabetes - DSM</title>
    <dc:date>2022-12-20T19:50:31+00:00</dc:date>
    <link>https://www.diabetesselfmanagement.com/news-research/2022/12/14/a1c-testing-may-help-detect-undiagnosed-diabetes/</link>
    <dc:creator>Michael.Massing</dc:creator><description><![CDATA["The researchers also found that using just age or body-mass index (BMI) to decide who should get screening for diabetes — and receive an A1C test — would miss more than 40% of undiagnosed diabetes cases."
[Validated risk scores] did better, with the ADA Risk Score missing 23.3% of undiagnosed diabetes cases, the Leicester Risk Score missing 15.7% of undiagnosed cases, and the Finnish Diabetes Risk Score missing 7.8%...when the lowest potential cutoff score was used.]]></description>
<dc:subject>diabetes diagnosis screening A1c BMI age risk validated score undiagnosed</dc:subject>
<dc:source>https://pinboard.in/</dc:source>
<dc:identifier>https://pinboard.in/u:Michael.Massing/b:9c5a8ebc62b3/</dc:identifier>
<taxo:topics><rdf:Bag>	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:diabetes"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:diagnosis"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:screening"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:A1c"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:BMI"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:age"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:risk"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:validated"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:score"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:undiagnosed"/>
</rdf:Bag></taxo:topics>
</item>
<item rdf:about="https://www.webmd.com/diet/ss/slideshow-zinc-mineral?ecd=wnl_spr_042122_remail&amp;ctr=wnl-spr-042122-remail_lead_cta&amp;mb=WlcCxCEv%408g239bDT2bGdeHnVev1imbCLJvzB2ihRmM%3D">
    <title>Why You Need Zinc and How to Get It</title>
    <dc:date>2022-10-04T04:02:48+00:00</dc:date>
    <link>https://www.webmd.com/diet/ss/slideshow-zinc-mineral?ecd=wnl_spr_042122_remail&amp;ctr=wnl-spr-042122-remail_lead_cta&amp;mb=WlcCxCEv%408g239bDT2bGdeHnVev1imbCLJvzB2ihRmM%3D</link>
    <dc:creator>Michael.Massing</dc:creator><description><![CDATA[Too Much Can Be Bad for You
14/15
It can cause diarrhea, stomach cramps, headache, and nausea. And if you take too much for too long, you may have lower levels of copper (another essential nutrient), a weaker immune system, and less HDL -- or “good” -- cholesterol. You shouldn’t get more than 40 milligrams a day unless your doctor has told you otherwise.
Interactions With Other Medication
15/15
Zinc supplements can weaken the effects of antibiotics, and antibiotics can make it harder for your body to use zinc. The supplements also can make it harder for your body to absorb some drugs, like the arthritis drug penicillamine. ]]></description>
<dc:subject>Zinc food foods supplements risk benefit</dc:subject>
<dc:source>https://pinboard.in/</dc:source>
<dc:identifier>https://pinboard.in/u:Michael.Massing/b:a342ba304879/</dc:identifier>
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	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:food"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:foods"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:supplements"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:risk"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:benefit"/>
</rdf:Bag></taxo:topics>
</item>
<item rdf:about="https://www.medpagetoday.com/opinion/second-opinions/100409?xid=nl_secondopinion_2022-08-28&amp;eun=g894600d0r">
    <title>The Burden of Obesity Is Not Carried Equally | MedPage Today</title>
    <dc:date>2022-08-28T19:13:48+00:00</dc:date>
    <link>https://www.medpagetoday.com/opinion/second-opinions/100409?xid=nl_secondopinion_2022-08-28&amp;eun=g894600d0r</link>
    <dc:creator>Michael.Massing</dc:creator><description><![CDATA[Despite researchers making significant advances in the last 2 decades, obesity is too often myopically viewed as the result of an individual's lifestyle choices around diet and exercise. Viewing the disease through this lens omits that body weight is determined by a combination of genetic, metabolic, behavioral, environmental, cultural, and socioeconomic factors. In fact, we know that a significant proportion of obesity can be influenced by genetics.]]></description>
<dc:subject>obesity health correlation comorbidities risk population race racial ethnic disparities gender sex care malnutrition</dc:subject>
<dc:source>https://pinboard.in/</dc:source>
<dc:identifier>https://pinboard.in/u:Michael.Massing/b:f09dc7f82dea/</dc:identifier>
<taxo:topics><rdf:Bag>	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:obesity"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:health"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:correlation"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:comorbidities"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:risk"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:population"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:race"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:racial"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:ethnic"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:disparities"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:gender"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:sex"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:care"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:malnutrition"/>
</rdf:Bag></taxo:topics>
</item>
<item rdf:about="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6587545/">
    <title>Worsening of diabetic retinopathy with rapid improvement in systemic glucose control: A review - PMC</title>
    <dc:date>2022-04-08T02:24:59+00:00</dc:date>
    <link>https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6587545/</link>
    <dc:creator>Michael.Massing</dc:creator><dc:subject>deterioration worsening diabetic retinopathy risk benefit human in vivo research peer-reviewed rapid improvement glucose control A1c review</dc:subject>
<dc:source>https://pinboard.in/</dc:source>
<dc:identifier>https://pinboard.in/u:Michael.Massing/b:4109b06e318f/</dc:identifier>
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	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:A1c"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:review"/>
</rdf:Bag></taxo:topics>
</item>
<item rdf:about="https://www.medpagetoday.com/neurology/dementia/97854?xid=nl_covidupdate_2022-03-25&amp;eun=g894600d0r">
    <title>Blood Profile at Age 35 Linked to Subsequent Alzheimer's Dementia | MedPage Today</title>
    <dc:date>2022-03-26T18:35:18+00:00</dc:date>
    <link>https://www.medpagetoday.com/neurology/dementia/97854?xid=nl_covidupdate_2022-03-25&amp;eun=g894600d0r</link>
    <dc:creator>Michael.Massing</dc:creator><description><![CDATA[High-density lipoprotein (HDL) cholesterol and triglyceride levels in people as young as age 35 were linked with Alzheimer's dementia decades later in life, longitudinal data from the Framingham Heart Study showed.

Risk of Alzheimer's dementia fell by 15.4% during early adulthood (ages 35 to 50) and by 17.8% during middle adulthood (ages 51 to 60) for every 15 mg/dL increase in HDL cholesterol, reported Xiaoling Zhang, MD, PhD, of Boston University School of Medicine, and co-authors in Alzheimer's & Dementia.

A 15 mg/dL increase in blood glucose measured during middle adulthood was associated with a 14.5% increased Alzheimer's risk, they added. Triglyceride levels were associated with Alzheimer's only in the early adulthood group. Findings remained significant after adjusting for treatment.

"These findings show for the first time that cardiovascular risk factors, including HDL, which has not been consistently reported as a strong risk factor for Alzheimer's disease, contribute to future risk of Alzheimer's disease starting as early as age 35," Zhang said in a statement.

Other vascular risk factors, including low-density lipoprotein (LDL) cholesterol, total cholesterol, BMI, smoking, and systolic blood pressure, were not associated with Alzheimer's dementia in any stage of adulthood (P>0.05).

Future development of Alzheimer's dementia was progressively higher and likely to occur earlier among people who had blood glucose in pre-diabetic (100 to 126 mg/dL) and diabetic (>126 mg/dL) ranges in early adulthood and middle adulthood.

"Intervention targeting cholesterol and glucose management starting in early adulthood can help maximize cognitive health in later life," Farrer suggested.]]></description>
<dc:subject>Framingham longitudinal data risk Alzheimer's cardiovascular factors peer-reviewed research HDL high-density lipoprotein correlation cohort human in vivo prospective trial prevention intervention</dc:subject>
<dc:source>https://pinboard.in/</dc:source>
<dc:identifier>https://pinboard.in/u:Michael.Massing/b:1b2481ed6811/</dc:identifier>
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	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:longitudinal"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:data"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:risk"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:Alzheimer's"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:cardiovascular"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:factors"/>
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	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:lipoprotein"/>
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	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:prospective"/>
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	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:intervention"/>
</rdf:Bag></taxo:topics>
</item>
<item rdf:about="https://academic.oup.com/eurjpc/advance-article/doi/10.1093/eurjpc/zwab189/6447088">
    <title>Sodium glucose co-transporter 2 inhibitors in heart failure with preserved ejection fraction: a systematic review and meta-analysis | European Journal of Preventive Cardiology | Oxford Academic</title>
    <dc:date>2021-12-16T03:23:08+00:00</dc:date>
    <link>https://academic.oup.com/eurjpc/advance-article/doi/10.1093/eurjpc/zwab189/6447088</link>
    <dc:creator>Michael.Massing</dc:creator><description><![CDATA[We confirm that the use of SGLT2i is associated with a substantial decrease in the risk of CV death or HHF in patients with heart failure and ejection fraction >40%. Importantly, in this first and largest meta-analysis reviewing LVEF >50%, we show that this benefit is maintained, albeit to a lesser degree, in the cohort of patients with LVEF ≥50%. SGLT2i become in this way the first medication with potential for prognostic benefit in HFpEF. However, overall mortality was not improved in HFpEF, indicating that the other comorbidities associated with HFpEF play a significant role. In order to draw robust conclusions safely regarding these efficacy outcomes, further large trials need to evaluate the effect of the SGLT2 inhibitors in a sufficient number of patients with HFpEF.

The various subphenotypes of HFpEF and its management have puzzled physicians for several years. The high rates of morbidity and mortality that it carries, along with the diagnostic and treatment challenges, have transformed it in one of the most challenging clinical entities. SGLT2i appear to provide some light and positivity, as their cardiometabolic profile impacts favourably on the complex pathophysiological mechanisms involved in HFpEF.9 However, the quest to untangle the complexity of treating HFpEF is certainly not over. We show conclusively that for LVEF 40–50% included as HFpEF in previous studies (but no longer considered HFpEF in the guidelines) there is a strong benefit from SGLT2i translating to reduced cardiovascular mortality and HHF, but not overall mortality. This also extended to the LVEF >50% but with a smaller effect. Additionally, while the vast majority of the patients included in this meta-analysis were individuals with diabetes at baseline, the cardioprotective benefits of the SGLT2i have been shown to occur via mechanisms independent of baseline diabetes status.10 Furthermore, the effect of SGLT2i in patients with reduced EF without diabetes is well-documented9,10 and EMPEROR-Preserved also included patients without diabetes, therefore their effect is applicable to patients without diabetes.]]></description>
<dc:subject>SGLT2 inhibitor risk benefit heart failure ejection fraction meta-analysis diabetes comorbidity cardiac cardiovascular protection peer-reviewed research</dc:subject>
<dc:source>https://pinboard.in/</dc:source>
<dc:identifier>https://pinboard.in/u:Michael.Massing/b:c3be4a0be53b/</dc:identifier>
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	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:failure"/>
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	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:meta-analysis"/>
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</item>
<item rdf:about="https://khn.org/news/article/rural-hospital-closures-georgia/">
    <title>A Rural Georgia Community Reels After Its Hospital Closes | Kaiser Health News</title>
    <dc:date>2021-12-15T15:51:01+00:00</dc:date>
    <link>https://khn.org/news/article/rural-hospital-closures-georgia/</link>
    <dc:creator>Michael.Massing</dc:creator><dc:subject>Medicaid expansion morbidity mortality risk obstructionism diagnosis treatment health disparities cost benefit poverty SDH social determinants of epidemiology correlation chronic population obstruction public disease burden access care healthcare rural hospitals research data sources</dc:subject>
<dc:source>https://pinboard.in/</dc:source>
<dc:identifier>https://pinboard.in/u:Michael.Massing/b:582a9185ac41/</dc:identifier>
<taxo:topics><rdf:Bag>	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:Medicaid"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:expansion"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:morbidity"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:mortality"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:risk"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:obstructionism"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:diagnosis"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:treatment"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:health"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:disparities"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:cost"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:benefit"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:poverty"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:SDH"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:social"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:determinants"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:of"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:epidemiology"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:correlation"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:chronic"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:population"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:obstruction"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:public"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:disease"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:burden"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:access"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:care"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:healthcare"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:rural"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:hospitals"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:research"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:data"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:sources"/>
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</item>
<item rdf:about="https://www.medpagetoday.com/primarycare/sleepdisorders/96030?xid=nl_mpt_DHE_2021-12-12&amp;eun=g894600d0r">
    <title>Cannabis Not So Great for Catching Z's | MedPage Today</title>
    <dc:date>2021-12-13T02:46:13+00:00</dc:date>
    <link>https://www.medpagetoday.com/primarycare/sleepdisorders/96030?xid=nl_mpt_DHE_2021-12-12&amp;eun=g894600d0r</link>
    <dc:creator>Michael.Massing</dc:creator><dc:subject>cannabis sleep risk benefit THC CBD obstructive apnea OSA peer-reviewed research human trial retrospective recall</dc:subject>
<dc:source>https://pinboard.in/</dc:source>
<dc:identifier>https://pinboard.in/u:Michael.Massing/b:3198bd478de6/</dc:identifier>
<taxo:topics><rdf:Bag>	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:cannabis"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:sleep"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:risk"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:benefit"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:THC"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:CBD"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:obstructive"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:apnea"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:OSA"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:peer-reviewed"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:research"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:human"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:trial"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:retrospective"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:recall"/>
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</item>
<item rdf:about="https://academic.oup.com/ajcn/article/93/5/1062/4597729">
    <title>High-protein, reduced-carbohydrate weight-loss diets promote metabolite profiles likely to be detrimental to colonic health | The American Journal of Clinical Nutrition | Oxford Academic</title>
    <dc:date>2021-12-01T04:12:01+00:00</dc:date>
    <link>https://academic.oup.com/ajcn/article/93/5/1062/4597729</link>
    <dc:creator>Michael.Massing</dc:creator><description><![CDATA[We designed this study to assess the effect of diets with reduced carbohydrate and increased protein contents on metabolites considered to influence long-term colonic health, in particular the risk of colorectal disease.

Design: We provided 17 obese men with a defined weight-maintenance diet (85 g protein, 116 g fat, and 360 g carbohydrate/d) for 7 d followed by 4 wk each of a high-protein and moderate-carbohydrate (HPMC; 139 g protein, 82 g fat, and 181 g carbohydrate/d) diet and a high-protein and low-carbohydrate (HPLC; 137 g protein, 143 g fat, and 22 g carbohydrate/d) diet in a crossover design. Fecal samples were analyzed to determine concentrations of phenolic metabolites, short-chain fatty acids, and nitrogenous compounds of dietary and microbial origin.

Results: Compared with the maintenance diet, the HPMC and HPLC diets resulted in increased proportions of branched-chain fatty acids and concentrations of phenylacetic acid and N-nitroso compounds. The HPLC diet also decreased the proportion of butyrate in fecal short-chain fatty acid concentrations, which was concomitant with a reduction in the Roseburia/Eubacterium rectale group of bacteria, and greatly reduced concentrations of fiber-derived, antioxidant phenolic acids such as ferulate and its derivatives.

Conclusions: After 4 wk, weight-loss diets that were high in protein but reduced in total carbohydrates and fiber resulted in a significant decrease in fecal cancer-protective metabolites and increased concentrations of hazardous metabolites. Long-term adherence to such diets may increase risk of colonic disease.]]></description>
<dc:subject>diet study clinical research in vivo human low carb carbohydrate high protein weight fat loss maintenance benefit risk keto peer-reviewed crossover</dc:subject>
<dc:source>https://pinboard.in/</dc:source>
<dc:identifier>https://pinboard.in/u:Michael.Massing/b:c7ae6abdda0e/</dc:identifier>
<taxo:topics><rdf:Bag>	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:diet"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:study"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:clinical"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:research"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:in"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:vivo"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:human"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:low"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:carb"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:carbohydrate"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:high"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:protein"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:weight"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:fat"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:loss"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:maintenance"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:benefit"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:risk"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:keto"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:peer-reviewed"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:crossover"/>
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</item>
<item rdf:about="https://www.thelancet.com/journals/lancet/article/PIIS0140-6736(21)02188-7/fulltext">
    <title>Tirzepatide versus insulin glargine in type 2 diabetes and increased cardiovascular risk (SURPASS-4): a randomised, open-label, parallel-group, multicentre, phase 3 trial - The Lancet</title>
    <dc:date>2021-10-25T01:20:04+00:00</dc:date>
    <link>https://www.thelancet.com/journals/lancet/article/PIIS0140-6736(21)02188-7/fulltext</link>
    <dc:creator>Michael.Massing</dc:creator><description><![CDATA[[Lilly assesses] efficacy and [especially cardiovascular] safety of the novel dual GIP and GLP-1 receptor agonist tirzepatide vs insulin glargine in adult [type 2 diabetics with high CV] risk inadequately controlled on oral glucose-lowering medications.
]]></description>
<dc:subject>insulin glargine GIP GLP-1 receptor A1c Hb hemoglobin benefit risk cardiovascular peer-reviewed research clinical trial human in vivo</dc:subject>
<dc:source>https://pinboard.in/</dc:source>
<dc:identifier>https://pinboard.in/u:Michael.Massing/b:eb3838f4fd08/</dc:identifier>
<taxo:topics><rdf:Bag>	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:insulin"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:glargine"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:GIP"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:GLP-1"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:receptor"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:A1c"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:Hb"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:hemoglobin"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:benefit"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:risk"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:cardiovascular"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:peer-reviewed"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:research"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:clinical"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:trial"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:human"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:in"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:vivo"/>
</rdf:Bag></taxo:topics>
</item>
<item rdf:about="https://heart.bmj.com/content/early/2021/10/13/heartjnl-2021-319672">
    <title>Anticoagulant prescribing for atrial fibrillation and risk of incident dementia | Heart</title>
    <dc:date>2021-10-24T22:37:12+00:00</dc:date>
    <link>https://heart.bmj.com/content/early/2021/10/13/heartjnl-2021-319672</link>
    <dc:creator>Michael.Massing</dc:creator><description><![CDATA[The aim of this study was to investigate the association between oral anticoagulant type (direct oral anticoagulants (DOACs) vs vitamin K antagonists (VKAs)) and incident dementia or mild cognitive impairment (MCI) among patients with newly diagnosed atrial fibrillation (AF).

Methods Using linked electronic health record (EHR) data from the Clinical Practice Research Datalink in the UK, we conducted a historical cohort study among first-time oral anticoagulant users with incident non-valvular AF diagnosed from 2012 to 2018. We compared the incidence of (1) clinically coded dementia and (2) MCI between patients prescribed VKAs and DOACs using Cox proportional hazards regression models, with age as the underlying timescale, accounting for calendar time and time on treatment, sociodemographic and lifestyle factors, clinical comorbidities and medications.

Results Of 39 200 first-time oral anticoagulant users (44.6% female, median age 76 years, IQR 68–83), 20 687 (53%) were prescribed a VKA and 18 513 (47%) a DOAC at baseline. Overall, 1258 patients (3.2%) had GP-recorded incident dementia, incidence rate 16.5 per 1000 person-years. DOAC treatment for AF was associated with a 16% reduction in dementia diagnosis compared with VKA treatment in the whole cohort (adjusted HR 0.84, 95% CI: 0.73 to 0.98) and with a 26% reduction in incident MCI (adjusted HR 0.74, 95% CI: 0.65 to 0.84). Findings were similar across various sensitivity analyses.

Conclusions Incident EHR-recorded dementia and MCI were less common among patients prescribed DOACs for new AF compared with those prescribed VKAs.]]></description>
<dc:subject>atrial fibrillation treatment options risk benefit cardiovasculr cohort study peer-reviewed research human retrospective</dc:subject>
<dc:source>https://pinboard.in/</dc:source>
<dc:identifier>https://pinboard.in/u:Michael.Massing/b:2670b7dfdf20/</dc:identifier>
<taxo:topics><rdf:Bag>	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:atrial"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:fibrillation"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:treatment"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:options"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:risk"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:benefit"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:cardiovasculr"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:cohort"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:study"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:peer-reviewed"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:research"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:human"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:retrospective"/>
</rdf:Bag></taxo:topics>
</item>
<item rdf:about="https://www.mdpi.com/2072-6643/13/10/3299/htm#B94-nutrients-13-03299">
    <title>Nutrients | Free Full-Text | Alternative Dietary Patterns for Americans: Low-Carbohydrate Diets | HTML</title>
    <dc:date>2021-10-06T03:12:26+00:00</dc:date>
    <link>https://www.mdpi.com/2072-6643/13/10/3299/htm#B94-nutrients-13-03299</link>
    <dc:creator>Michael.Massing</dc:creator><description><![CDATA[Data Availability Statement
No original data were generated for this manuscript.

Conflicts of Interest
JSV receives royalties for low-carbohydrate nutrition books, is co-founder and has equity in Virta Health Corp, and is a scientific advisor to Simply Good Foods; SDP receives royalties for low-carbohydrate nutrition books and is co-founder and has equity in Virta Health Corp; RMK is on the scientific advisory boards of Virta Health Corp, Seraphina Therapeutics, and Day Two, and has licensed patents for lipoprotein particle analysis; RJJ has received honoraria from Horizon Pharma and Danone, has stocks with XORTX Therapeutics, and has equity with Colorado Partners LLC (CRP); LRS is a consultant with Sentinel Management; WSY is a consultant for Guideline Central and dietdoctor.com; NT receives book royalties and honoraria for speeches via the Harry Walker Agency.

The decades-long dietary experiment embodied in the Dietary Guidelines for Americans (DGA) focused on limiting fat, especially saturated fat, and higher carbohydrate intake has coincided with rapidly escalating epidemics of obesity and type 2 diabetes (T2D) that are contributing to the progression of cardiovascular disease (CVD) and other diet-related chronic diseases. Moreover, the lack of flexibility in the DGA as it pertains to low carbohydrate approaches does not align with the contemporary trend toward precision nutrition. We argue that personalizing the level of dietary carbohydrate should be a high priority based on evidence that Americans have a wide spectrum of metabolic variability in their tolerance to high carbohydrate loads. Obesity, metabolic syndrome, and T2D are conditions strongly associated with insulin resistance, a condition exacerbated by increased dietary carbohydrate and improved by restricting carbohydrate. Low-carbohydrate diets are grounded across the time-span of human evolution, have well-established biochemical principles, and are now supported by multiple clinical trials in humans that demonstrate consistent improvements in multiple established risk factors associated with insulin resistance and cardiovascular disease. The American Diabetes Association (ADA) recently recognized a low carbohydrate eating pattern as an effective approach for patients with diabetes. Despite this evidence base, low-carbohydrate diets are not reflected in the DGA. As the DGA Dietary Patterns have not been demonstrated to be universally effective in addressing the needs of many Americans and recognizing the lack of widely available treatments for obesity, metabolic syndrome, and T2D that are safe, effective, and sustainable, the argument for an alternative, low-carbohydrate Dietary Pattern is all the more compelling....
Recently, the American Diabetes Association (ADA) has updated its nutrition recommendations to allow for more flexibility. Starting with their 2019 standards of care for patients with diabetes, the ADA stated that “Low-carbohydrate eating patterns, especially very low-carbohydrate (VLC) eating patterns, have been shown to reduce A1C and the need for antihyperglycemic medications. These eating patterns are among the most studied eating patterns for type 2 diabetes.”]]></description>
<dc:subject>low carbohydrate diet carb dietary guidelines for Americans DGA insulin resistance metabolic syndrome risk benefit peer-reviewed research</dc:subject>
<dc:source>https://pinboard.in/</dc:source>
<dc:identifier>https://pinboard.in/u:Michael.Massing/b:9aabcf072568/</dc:identifier>
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	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:diet"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:carb"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:dietary"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:guidelines"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:for"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:Americans"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:DGA"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:insulin"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:resistance"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:metabolic"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:syndrome"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:risk"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:benefit"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:peer-reviewed"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:research"/>
</rdf:Bag></taxo:topics>
</item>
<item rdf:about="https://www.uptodate.com/contents/pneumococcal-vaccination-in-adults">
    <title>Pneumococcal vaccination in adults - UpToDate</title>
    <dc:date>2021-09-26T02:36:30+00:00</dc:date>
    <link>https://www.uptodate.com/contents/pneumococcal-vaccination-in-adults</link>
    <dc:creator>Michael.Massing</dc:creator><description><![CDATA[Adults, ages 65 years or older — Vaccination with PPSV23 is indicated for all adults because the overall incidence of pneumococcal disease rises greatly after this age (table 3) [18,20]. However, the incidence of pneumococcal disease specifically caused by PCV13 serotypes is low as a result of widespread childhood vaccination (figure 2) [8]; thus:

●We do not routinely vaccinate adults ≥65 years old with PCV13 unless they have another indication for PCV13 (ie, immunocompromise, asplenia, cerebrospinal fluid leak, cochlear implant, or history invasive pneumococcal disease) and have not previously received the vaccine.
●For adults with certain chronic conditions that place them at higher risk for pneumococcal disease (ie, chronic cardiac, lung, and/or liver disease, diabetes mellitus, smoking, and/or alcohol use disorder), we discuss risks and benefits of PCV13 on an individual basis when deciding to give this vaccine. For most persons in the United States, we consider the absolute risk of acquiring pneumococcal disease caused by a PCV13 serotype to be so low that the expected benefit is very small.
The ACIP has rescinded its prior recommendation to vaccinate all adults ≥65 years old with both PCV13 and PPSV23 [23]. The committee now recommends PPSV23 for all patients in this age group and advises clinicians to engage in shared decision-making to determine whether PCV13 should be given in addition to PPSV23 for patients who otherwise lack an indication for dual vaccination (eg, asplenia, cochlear implant) (table 4) [20]. The change in recommendation is based on the dramatic reduction in the incidence of infection with PCV13 serotypes that has resulted from universal childhood PCV13 vaccination [14]. Because the likelihood of infection with a PCV13 serotype is now so low, we generally find that the benefits of the additional vaccination with PCV13 do not outweigh its risk or costs.]]></description>
<dc:subject>pneumonia vaccination pneumococcal disease aging risk benefit prevalence incidence guidelines peer-reviewed research overview human in vivo</dc:subject>
<dc:source>https://pinboard.in/</dc:source>
<dc:identifier>https://pinboard.in/u:Michael.Massing/b:3c489e81785e/</dc:identifier>
<taxo:topics><rdf:Bag>	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:pneumonia"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:vaccination"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:pneumococcal"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:disease"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:aging"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:risk"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:benefit"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:prevalence"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:incidence"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:guidelines"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:peer-reviewed"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:research"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:overview"/>
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	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:in"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:vivo"/>
</rdf:Bag></taxo:topics>
</item>
<item rdf:about="https://www.endocrinologyadvisor.com/home/topics/diabetes/increased-risk-for-major-adverse-cardiovascular-events-with-insulin-in-patients-with-diabetes-and-acs/?NID=-1&amp;cpn&amp;email_hash=983917dd1eba78eb44e393fea65df5ca&amp;hmEmail=Jw_YJjhVyI84iX-Oepu7iw7YiNSIB0EK31LP7LVx8w01&amp;hmSubId=YY-zPItNrOw1&amp;mpweb=1323-153862-7624403">
    <title>Insulin Use Linked to Risk for MACE Among Individuals With Stable Diabetes and Acute Coronary Syndrome - Endocrinology Advisor</title>
    <dc:date>2021-09-21T02:51:49+00:00</dc:date>
    <link>https://www.endocrinologyadvisor.com/home/topics/diabetes/increased-risk-for-major-adverse-cardiovascular-events-with-insulin-in-patients-with-diabetes-and-acs/?NID=-1&amp;cpn&amp;email_hash=983917dd1eba78eb44e393fea65df5ca&amp;hmEmail=Jw_YJjhVyI84iX-Oepu7iw7YiNSIB0EK31LP7LVx8w01&amp;hmSubId=YY-zPItNrOw1&amp;mpweb=1323-153862-7624403</link>
    <dc:creator>Michael.Massing</dc:creator><dc:subject>treatment risk benefit insulin cardiovascular heart ACS acute coronary syndome peer-reviewed research in vivo human clinical trial</dc:subject>
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<item rdf:about="https://www.diabetesprevention.pitt.edu/for-the-public/diabetes-prevention-program-dpp/">
    <title>Diabetes Prevention Program (DPP)</title>
    <dc:date>2021-09-16T22:50:01+00:00</dc:date>
    <link>https://www.diabetesprevention.pitt.edu/for-the-public/diabetes-prevention-program-dpp/</link>
    <dc:creator>Michael.Massing</dc:creator><description><![CDATA[The DPP’s striking results tell us that diet, exercise, and behavior modification, delays and may prevent the development of type 2 diabetes. The DPP also suggests that Metformin is effective in delaying (or preventing) the onset of diabetes.

Participants in the lifestyle intervention group–those receiving intensive counseling on effective diet, exercise, and behavior modification–reduced their risk of developing diabetes by 58%. This finding was true across all participating ethnic groups and for both men and women. Lifestyle changes worked particularly well for participants aged 60 and older, reducing their risk by 71%. About 5% of the lifestyle intervention group developed diabetes each year during the study period, compared with 11% in those who did not get the intervention. Researchers think that weight loss–achieved through better eating habits and exercise–reduces the risk of diabetes by improving the body’s ability to use insulin and process glucose.

Participants taking Metformin reduced their risk of developing diabetes by 31%. Metformin was effective for both men and women, but it was less effective in people aged 45 and older. Metformin was most effective in people 25 to 44 years old and in those with a body mass index of 35 or higher (at least 60 pounds overweight). About 7.8% of the Metformin group developed diabetes each year during the study, compared with 11% of the group receiving the placebo.

NIH Publication No. 06–5099
August 2006]]></description>
<dc:subject>diabetes type 2 T2D prevention intervention behavioral diet exercise activity weight fat loss body risk factors in vivo human clinical trial situ support treatment progression management resource information education</dc:subject>
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<item rdf:about="https://www.endocrinologyadvisor.com/home/topics/diabetes/type-2-diabetes/weight-loss-and-cardiovascular-renal-benefit-with-sglt2-inhibitors-in-t2d/?hmSubId=YY-zPItNrOw1&amp;hmEmail=Jw_YJjhVyI84iX-Oepu7iw7YiNSIB0EK31LP7LVx8w01&amp;NID=-1&amp;email_hash=983917dd1eba78eb44e393fea65df5ca&amp;mpweb=1323-149900-7624403">
    <title>SGLT2 Inhibitors Lead to Favorable Renal, CV Outcomes in Type 2 Diabetes - Endocrinology Advisor</title>
    <dc:date>2021-08-25T17:28:16+00:00</dc:date>
    <link>https://www.endocrinologyadvisor.com/home/topics/diabetes/type-2-diabetes/weight-loss-and-cardiovascular-renal-benefit-with-sglt2-inhibitors-in-t2d/?hmSubId=YY-zPItNrOw1&amp;hmEmail=Jw_YJjhVyI84iX-Oepu7iw7YiNSIB0EK31LP7LVx8w01&amp;NID=-1&amp;email_hash=983917dd1eba78eb44e393fea65df5ca&amp;mpweb=1323-149900-7624403</link>
    <dc:creator>Michael.Massing</dc:creator><description><![CDATA[There was a “possible benefit” for SGLT2 inhibitors in regard to major adverse CV events across 4 systematic reviews (OR/HR range, 0.80-0.89). In contrast, SGLT2 inhibitors featured “clear evidence of no effect or equivalence” for stroke and fractures. Also, there was a “clear evidence of harm” with SGLT2 inhibitors for genital infections (RR/OR range, 2.06-5.25) and ketoacidosis (HR/OR range, 1.36-2.20).]]></description>
<dc:subject>SGLT2 inhibitors peer-reviewed research human clinical trial review in vivo situ risk benefit diabetes treatment T2D type 2 kidney heart cardiovascular CVD genital infection necrosis diabetic ketoacidosis DKA</dc:subject>
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<item rdf:about="https://www.diabetes.org/a1c/diagnosis">
    <title>Diagnosis | ADA</title>
    <dc:date>2021-06-15T02:35:07+00:00</dc:date>
    <link>https://www.diabetes.org/a1c/diagnosis</link>
    <dc:creator>Michael.Massing</dc:creator><dc:subject>diabetes diagnostic diagnosis standard threshold glycated hemoglobin A1c HbA1c prediabetes normal risk blood glucose test random fasting sugar oral tolerance ADA</dc:subject>
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<item rdf:about="https://www.mayoclinic.org/diseases-conditions/diabetes/diagnosis-treatment/drc-20371451">
    <title>Diabetes - Diagnosis and treatment - Mayo Clinic</title>
    <dc:date>2021-06-15T02:11:56+00:00</dc:date>
    <link>https://www.mayoclinic.org/diseases-conditions/diabetes/diagnosis-treatment/drc-20371451</link>
    <dc:creator>Michael.Massing</dc:creator><description><![CDATA[Glycated hemoglobin (A1C) test.
An A1C level of 6.5% or higher on two separate tests indicates that you have diabetes. An A1C between 5.7 and 6.4 % indicates prediabetes. Below 5.7 is considered normal.

Random blood sugar test. 
A blood sample will be taken at a random time. Regardless of when you last ate, a blood sugar level of 200 milligrams per deciliter (mg/dL) — 11.1 millimoles per liter (mmol/L) — or higher suggests diabetes.

Fasting blood sugar test. 
A blood sample will be taken after an overnight fast. A fasting blood sugar level less than 100 mg/dL (5.6 mmol/L) is normal. A fasting blood sugar level from 100 to 125 mg/dL (5.6 to 6.9 mmol/L) is considered prediabetes. If it's 126 mg/dL (7 mmol/L) or higher on two separate tests, you have diabetes.

Oral glucose tolerance test. For this test, you fast overnight, and the fasting blood sugar level is measured. Then you drink a sugary liquid, and blood sugar levels are tested periodically for the next two hours.
A blood sugar level less than 140 mg/dL (7.8 mmol/L) is normal. A reading of more than 200 mg/dL (11.1 mmol/L) after two hours indicates diabetes. A reading between 140 and 199 mg/dL (7.8 mmol/L and 11.0 mmol/L) indicates prediabetes.]]></description>
<dc:subject>diabetes diagnostic diagnosis standard threshold glycated hemoglobin A1c HbA1c prediabetes normal risk blood glucose test random fasting sugar oral tolerance Mayo Clinic</dc:subject>
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<item rdf:about="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5664031/">
    <title>Curcumin: A Review of Its’ Effects on Human Health</title>
    <dc:date>2021-05-29T00:15:11+00:00</dc:date>
    <link>https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5664031/</link>
    <dc:creator>Michael.Massing</dc:creator><description><![CDATA[Curcumin has a long established safety record. For example, according to JECFA (The Joint United Nations and World Health Organization Expert Committee on Food Additives) and EFSA (European Food Safety Authority) reports, the Allowable Daily Intake (ADI) value of curcumin is 0–3 mg/kg body weight [65]. Several trials on healthy subjects have supported the safety and efficacy of curcumin. Despite this well-established safety, some negative side effects have been reported. Seven subjects receiving 500–12,000 mg in a dose response study and followed for 72 h experienced diarrhea, headache, rash, and yellow stool [19]. In another study, some subjects receiving 0.45 to 3.6 g/day curcumin for one to four months reported nausea and diarrhea and an increase in serum alkaline phosphatase and lactate dehydrogenase contents [66].

Go to:
7. Conclusions
Curcumin has received worldwide attention for its multiple health benefits, which appear to act primarily through its anti-oxidant and anti-inflammatory mechanisms. These benefits are best achieved when curcumin is combined with agents such as piperine, which increase its bioavailability significantly. Research suggests that curcumin can help in the management of oxidative and inflammatory conditions, metabolic syndrome, arthritis, anxiety, and hyperlipidemia. It may also help in the management of exercise-induced inflammation and muscle soreness, thus enhancing recovery and subsequent performance in active people. In addition, a relatively low dose can provide health benefits for people that do not have diagnosed health conditions.]]></description>
<dc:subject>turmeric piperine drug interacton elimination benefit risk potential overview supplement spices dose dosage adverse effects review peer-reviewed research clinical human in vivo</dc:subject>
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<item rdf:about="https://www.nbcnews.com/better/health/health-benefits-turmeric-it-s-complicated-ncna792901">
    <title>Is turmeric good for you? Yes, but It's complicated.</title>
    <dc:date>2021-05-29T00:11:46+00:00</dc:date>
    <link>https://www.nbcnews.com/better/health/health-benefits-turmeric-it-s-complicated-ncna792901</link>
    <dc:creator>Michael.Massing</dc:creator><description><![CDATA[f you’re bound and determined to experiment with curcumin medicinally, she recommends consulting your doctor — especially if you are already on medication — because preclinical studies have indicated it might change how other medications you use are metabolized in your body. That’s because curcumin supplements also an ingredient called piperine, which boosts the effects of curcumin but also its potential toxicity because it slows down the elimination of the curcumin and prescription drugs used for seizures, high blood pressure, angina and bipolar disorder.

To back her point that more research on people is needed, just this year, one 30-year old woman suffered a fatal outcome after receiving a turmeric-infused IV-drip holistic treatment. San Diego-area news outlet KGTV reported the woman died immediately from a heart attack after having the drip to treat her eczema.]]></description>
<dc:subject>turmeric piperine drug interacton elimination benefit risk potential overview supplement spices</dc:subject>
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<item rdf:about="https://www.mayoclinic.org/diseases-conditions/type-2-diabetes/expert-answers/blood-sugar/faq-20057941#:~:text=The%20average%20U.S.%20adult%20drinks,day%20appears%20to%20be%20safe.">
    <title>Caffeine: Does it affect blood sugar? - Mayo Clinic</title>
    <dc:date>2021-05-06T00:36:48+00:00</dc:date>
    <link>https://www.mayoclinic.org/diseases-conditions/type-2-diabetes/expert-answers/blood-sugar/faq-20057941#:~:text=The%20average%20U.S.%20adult%20drinks,day%20appears%20to%20be%20safe.</link>
    <dc:creator>Michael.Massing</dc:creator><description><![CDATA[The average U.S. adult drinks about two 8-ounce (240-milliliter) cups of coffee a day, which can contain around 280 milligrams of caffeine. For most young, healthy adults, caffeine doesn't appear to noticeably affect blood sugar (glucose) levels, and having up to 400 milligrams a day appears to be safe.

Some studies suggest that drinking coffee — whether caffeinated and decaffeinated — may actually reduce your risk of developing type 2 diabetes.

If you already have diabetes, however, the impact of caffeine on insulin action may be associated with higher or lower blood sugar levels. For some people with diabetes, about 200 milligrams of caffeine — or the equivalent of one to two 8-ounce (240-milliliter) cups of plain, brewed coffee — may cause this effect.

Caffeine affects every person differently. If you have diabetes or you're struggling to control your blood sugar levels, limiting the amount of caffeine in your diet may provide a benefit.]]></description>
<dc:subject>coffee caffeine blood sugar glucose fluctuation correlation variability sensitivity insulin overview risk benefit human research in vivo</dc:subject>
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<item rdf:about="https://www.webmd.com/diabetes/diabetes-and-caffeine">
    <title>Type 2 Diabetes and Caffeine: The Truth about Blood Sugar</title>
    <dc:date>2021-05-06T00:33:53+00:00</dc:date>
    <link>https://www.webmd.com/diabetes/diabetes-and-caffeine</link>
    <dc:creator>Michael.Massing</dc:creator><description><![CDATA[A growing body of research suggests people with type 2 diabetes react to caffeine differently. It can raise blood sugar and insulin levels for those with the disease.

One study looked at people with type 2 diabetes who took a 250-milligram caffeine pill at breakfast and another at lunchtime. That’s about the same amount as drinking two cups of coffee with each meal. The result: Their blood sugar was 8% higher than on days when they didn’t have caffeine. Their reading also jumped by more after each meal.

That’s because caffeine can affect how your body responds to insulin, the hormone that allows sugar to enter your cells and get changed into energy.


Caffeine may lower your insulin sensitivity. That means your cells don’t react to the hormone by as much as they once did. They don’t absorb as much sugar from your blood after you eat or drink. This causes your body to make more insulin, so you have higher levels after meals.]]></description>
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<item rdf:about="https://care.diabetesjournals.org/content/27/12/2990">
    <title>Effects of Coffee Consumption on Fasting Blood Glucose and Insulin Concentrations | Diabetes Care</title>
    <dc:date>2021-05-06T00:32:33+00:00</dc:date>
    <link>https://care.diabetesjournals.org/content/27/12/2990</link>
    <dc:creator>Michael.Massing</dc:creator><description><![CDATA[Higher habitual coffee consumption was associated with higher insulin sensitivity (1) and a lower risk for type 2 diabetes (2–6) in diverse populations. In contrast, short-term metabolic studies showed that caffeine intake can acutely lower insulin sensitivity (7–9) and increase glucose concentrations (10–15). ]]></description>
<dc:subject>coffee caffeine blood sugar glucose fluctuation correlation variability sensitivity insulin peer-reviewed research human in vivo randomized controlled trial RCT crossover risk benefit</dc:subject>
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<item rdf:about="https://www.healthline.com/health/coffee-s-effect-diabetes#other-benefits">
    <title>Coffee and Diabetes: Prevention, Effects on Glucose and Insulin, and More</title>
    <dc:date>2021-05-06T00:27:04+00:00</dc:date>
    <link>https://www.healthline.com/health/coffee-s-effect-diabetes#other-benefits</link>
    <dc:creator>Michael.Massing</dc:creator><description><![CDATA[There’s a clear difference in how people with diabetes and people without diabetes respond to coffee and caffeine. A 2008 study had habitual coffee drinkers with type 2 diabetes continuously monitor their blood sugar while doing daily activities.

During the day, it was shown that right after they drank coffee, their blood sugar would soar. Blood sugar was higher on days that they drank coffee than it was on days they didn’t.]]></description>
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<item rdf:about="https://www.everydayhealth.com/type-2-diabetes/symptoms/surprising-causes-of-blood-sugar-swings/">
    <title>10 Surprising Causes of Blood Sugar Swings You Probably Didn't Know | Everyday Health</title>
    <dc:date>2021-05-06T00:23:30+00:00</dc:date>
    <link>https://www.everydayhealth.com/type-2-diabetes/symptoms/surprising-causes-of-blood-sugar-swings/</link>
    <dc:creator>Michael.Massing</dc:creator><description><![CDATA[How much caffeine is safe if you’re at risk of diabetes or already have the disease is murky. While some research, like a review study published in June 2016 in the European Journal of Nutrition, found that healthy, regular coffee drinkers who consumed three to four cups of coffee per day saw a reduced risk of developing type 2, earlier research suggests that consuming too much caffeine may cause blood sugar levels to spike in those who already have the disease.

According to the Mayo Clinic, while consuming up to 400 milligrams (mg) of caffeine per day is safe for most people, in those with diabetes, the substance can affect how insulin behaves, leading to low or high blood sugar. You may notice blood sugar swings after drinking only two 8-ounce plain cups of brewed coffee.

The important thing to know is that everybody is different, and it’s important to monitor your blood glucose to observe how caffeine affects you. If you're a heavy consumer of caffeinated beverages, which include diet cola as well as coffee and tea, consider cutting back to see if your glucose control improves, says Bonsignore.]]></description>
<dc:subject>coffee caffeine blood sugar glucose fluctuation correlation risk benefit human research in vivo</dc:subject>
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<item rdf:about="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6544578/">
    <title>Effects of coffee consumption on glucose metabolism: A systematic review of clinical trials</title>
    <dc:date>2021-05-01T01:23:31+00:00</dc:date>
    <link>https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6544578/</link>
    <dc:creator>Michael.Massing</dc:creator><dc:subject>coffee glucose metabolism risk benefit systematic review in vivo clinical trials human peer-reviewed research caffeine insulin resistance sensitivity</dc:subject>
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<item rdf:about="https://scholar.google.com/scholar?hl=en&amp;as_sdt=0%2C7&amp;as_vis=1&amp;q=whole+grains+longevity+cardiovascular+cardiac+risk&amp;btnG=">
    <title>whole grains longevity cardiovascular cardiac risk - Google Scholar</title>
    <dc:date>2021-03-28T00:49:12+00:00</dc:date>
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    <dc:creator>Michael.Massing</dc:creator><description><![CDATA[whole grains longevity cardiovascular cardiac risk]]></description>
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<item rdf:about="https://www.aarp.org/caregiving/health/info-2021/nursing-homes-minorities-covid-deaths.html">
    <title>Nursing Homes With More Minorities Had More COVID Deaths</title>
    <dc:date>2021-02-13T21:44:33+00:00</dc:date>
    <link>https://www.aarp.org/caregiving/health/info-2021/nursing-homes-minorities-covid-deaths.html</link>
    <dc:creator>Michael.Massing</dc:creator><description><![CDATA[Black Americans are more commonly admitted to facilities with “lower nurse staffing ratios, more serious regulatory deficiencies, and a higher likelihood of being terminated from the Medicaid program,” researchers wrote in the study.

"Non-White residents are more likely to live in facilities that are larger, which creates more opportunity for viral transmission,” they continued. “COVID-19 is more prevalent in non-White communities, and community spread is a factor associated with cases and deaths in nursing homes.” Minority residents, they said, are “in the eye of that perfect storm."

In the United States, minority populations have been disproportionately affected by COVID-19, and nursing homes and other long-term care facilities, such as assisted living residences, account for 36 percent of the more than 475,000 U.S. COVID-19 fatalities. Less than 1 percent of the population lives in long-term care.... 

Efforts need to go beyond prioritizing the nursing home community for vaccinations and also include better education and outreach to minority populations on the safety and efficacy of the shots. “Their concerns are rooted in a history where they haven't been prioritized,” she observes. “In fact, it's been the opposite in many cases, and they've been ignored or mistreated. So we must listen to their questions — they're real and they're important.”]]></description>
<dc:subject>population institutionalization long term care race racism structural research human aging health healthcare prevention pandemic risk factors ethnicity Medicaid expansion obstruction obstructionism morbidity mortality</dc:subject>
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<item rdf:about="https://www.medpagetoday.com/infectiousdisease/covid19/90569">
    <title>Early Plasma Tx Promising in Adults 65+ With Milder COVID-19 | MedPage Today</title>
    <dc:date>2021-01-10T17:21:17+00:00</dc:date>
    <link>https://www.medpagetoday.com/infectiousdisease/covid19/90569</link>
    <dc:creator>Michael.Massing</dc:creator><description><![CDATA[[The] trial differed from others: it focused on older adults, who are most affected by the pandemic, and convalescent plasma was given "in a mild stage" with the aim of preventing progression.

"Our primary endpoint" -- severe respiratory illness -- "was an enrollment criterion in previous studies," the group noted.


Patients were enrolled in Argentina from June 4 to Oct. 25. They included patients ages 65-74 with at least one comorbidity, and patients ages 75 and older irrespective of pre-existing conditions. They had tested positive for SARS-CoV-2 and had symptoms including fever, unexplained sweating or chills and dry cough for less than 48 hours.

Severe respiratory disease was defined as respiration at 30 breaths per minute or more or oxygen saturation of less than 93% on ambient air. They were assessed from 12 hours after infusion to day 15 of trial participation.

Overall, 160 patients underwent randomization. Mean age was 77, and 62% were women. A little more than half were 75 or older. Most patients had pre-existing conditions, with over two-thirds of both groups being treated for hypertension.

An intention-to-treat analysis found severe respiratory disease developed in 13 of 80 patients in the intervention group and 25 of 80 in the placebo group, for a relative risk reduction of 48% and a number needed to treat of 7 to avert one episode of severe respiratory disease.]]></description>
<dc:subject>comorbidities risk factor age aging COVID19 treatment therapy convalescent plasma peer-reviewed research random placebo progression early intervention clinical trial in vivo human situ NNT reduction</dc:subject>
<dc:source>https://pinboard.in/</dc:source>
<dc:identifier>https://pinboard.in/u:Michael.Massing/b:1d73e3c064d3/</dc:identifier>
<taxo:topics><rdf:Bag>	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:comorbidities"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:risk"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:factor"/>
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	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:aging"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:COVID19"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:treatment"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:therapy"/>
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	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:research"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:random"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:placebo"/>
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	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:early"/>
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	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:situ"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:NNT"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:reduction"/>
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</item>
<item rdf:about="https://link.springer.com/article/10.1007/s00125-020-05253-x#Sec4">
    <title>An atlas on risk factors for type 2 diabetes: a wide-angled Mendelian randomisation study | SpringerLink</title>
    <dc:date>2020-10-15T06:30:28+00:00</dc:date>
    <link>https://link.springer.com/article/10.1007/s00125-020-05253-x#Sec4</link>
    <dc:creator>Michael.Massing</dc:creator><dc:subject>diabetes type 2 risk factors T2D correlation peer-reviewed research epidemiology etiology</dc:subject>
<dc:source>https://pinboard.in/</dc:source>
<dc:identifier>https://pinboard.in/u:Michael.Massing/b:5ebc949a473c/</dc:identifier>
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	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:2"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:risk"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:factors"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:T2D"/>
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</item>
<item rdf:about="https://care.diabetesjournals.org/content/28/11/2756">
    <title>Increased Mortality Risks of Pre-Diabetes (Impaired Fasting Glucose) in Taiwan | Diabetes Care</title>
    <dc:date>2020-09-27T17:44:12+00:00</dc:date>
    <link>https://care.diabetesjournals.org/content/28/11/2756</link>
    <dc:creator>Michael.Massing</dc:creator><description><![CDATA[RESULTS—FBG ≥110 mg/dl was associated with increased mortality risks for all causes, cardiovascular diseases (CVD), and diabetes. IFG, when defined as 110–125 mg/dl, was associated with a significant increase for CVD and/or diabetes mortality. These mortality risks remained elevated when known CVD risk factors were adjusted for. The IFG group shared risk factor characteristics more with the FBG ≥126 mg/dl group than with the FBG <110 mg/dl group. When IFG was defined as 100–125 mg/dl, the number of subjects quadrupled, but mortality risks diminished substantially because of the inclusion of 100–109 mg/dl group. The lowest FBG group, 50–75 mg/dl, had a significant 2-fold risk from all causes.

CONCLUSIONS—There was an overall J-shaped relationship between all-cause mortality and FBG. IFG, when defined as 110–125 mg/dl, is an independent risk factor and should be aggressively treated as a disease because its subsequent mortality risks for CVD and diabetes were significantly increased. The newly defined IFG at 100–125 mg/dl did not have the predictive power for later increases in CVD or diabetes mortality.]]></description>
<dc:subject>FBG IFG impaired fasting blood glucose mortality CVD cardiovascular disease risk threshold diagnostic standards prediabetes diabetes all-cause peer-reviewed research in vivo human cohort study</dc:subject>
<dc:source>https://pinboard.in/</dc:source>
<dc:identifier>https://pinboard.in/u:Michael.Massing/b:117b43629584/</dc:identifier>
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</item>
<item rdf:about="https://www.medpagetoday.org/infectiousdisease/uritheflu/88447?xid=nl_mpt_DHE_2020-09-04&amp;eun=g894600d0r&amp;vpass=1">
    <title>Link Between Influenza and Acute Heart Issues Confirmed by CDC Study | MedPage Today</title>
    <dc:date>2020-09-21T07:59:58+00:00</dc:date>
    <link>https://www.medpagetoday.org/infectiousdisease/uritheflu/88447?xid=nl_mpt_DHE_2020-09-04&amp;eun=g894600d0r&amp;vpass=1</link>
    <dc:creator>Michael.Massing</dc:creator><description><![CDATA[Among more than 80,000 adults hospitalized with laboratory-confirmed influenza, nearly 12% suffered an acute cardiovascular event, most commonly acute heart failure and acute ischemic heart disease, according to a cross-sectional study from CDC researchers.
Note that clinicians should be advised to encourage influenza vaccination, especially in those with underlying chronic conditions, to protect against acute cardiovascular events associated with influenza.]]></description>
<dc:subject>influenza flu cardiovascular risk events human retrospective in vivo cross-sectional peer-reviewed research</dc:subject>
<dc:source>https://pinboard.in/</dc:source>
<dc:identifier>https://pinboard.in/u:Michael.Massing/b:bfcd7be5b46b/</dc:identifier>
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	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:cardiovascular"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:risk"/>
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	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:human"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:retrospective"/>
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	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:research"/>
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</item>
<item rdf:about="https://academic.oup.com/eurheartj/article/41/32/3038/5901158">
    <title>COVID-19 is, in the end, an endothelial disease | European Heart Journal | Oxford Academic</title>
    <dc:date>2020-09-21T06:00:41+00:00</dc:date>
    <link>https://academic.oup.com/eurheartj/article/41/32/3038/5901158</link>
    <dc:creator>Michael.Massing</dc:creator><description><![CDATA["SARS-CoV-2 produces protean manifestations ranging from head to toe, wreaking seemingly indiscriminate havoc on multiple organ systems including the lungs, heart, brain, kidney and vasculature." (You know, right? But the best clinical abstract I've read.) ]]></description>
<dc:subject>COVID19 COVID endothelial cardiovascular heart brain kidneys risk morbidity mortality</dc:subject>
<dc:source>https://twitter.com/</dc:source>
<dc:identifier>https://pinboard.in/u:Michael.Massing/b:6e7a6923a604/</dc:identifier>
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	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:COVID"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:endothelial"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:cardiovascular"/>
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	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:brain"/>
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	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:morbidity"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:mortality"/>
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</item>
<item rdf:about="https://www.google.com/search?newwindow=1&amp;client=safari&amp;rls=en&amp;sxsrf=ALeKk00DhLsxFCDJMbebcbFW-TZc-k2Tzw%3A1600586672296&amp;ei=sANnX6_VEcmc_Qbzm5uoBw&amp;q=polio+scare+swimming+pools+1950s+50s+1956&amp;oq=polio+scare+swimming+pools+1950s+50s+1956&amp;gs_lcp=CgZwc3ktYWIQAzoECAAQRzoFCCEQoAFQ3OgFWJT5BWD3ggZoAHABeACAAboBiAG1BJIBAzMuMpgBAKABAaoBB2d3cy13aXrIAQjAAQE&amp;sclient=psy-ab&amp;ved=0ahUKEwiv7aKAmvfrAhVJTt8KHfPNBnUQ4dUDCAw&amp;uact=5">
    <title>polio scare swimming pools 1950s 50s 1956 - Google Search</title>
    <dc:date>2020-09-20T07:32:28+00:00</dc:date>
    <link>https://www.google.com/search?newwindow=1&amp;client=safari&amp;rls=en&amp;sxsrf=ALeKk00DhLsxFCDJMbebcbFW-TZc-k2Tzw%3A1600586672296&amp;ei=sANnX6_VEcmc_Qbzm5uoBw&amp;q=polio+scare+swimming+pools+1950s+50s+1956&amp;oq=polio+scare+swimming+pools+1950s+50s+1956&amp;gs_lcp=CgZwc3ktYWIQAzoECAAQRzoFCCEQoAFQ3OgFWJT5BWD3ggZoAHABeACAAboBiAG1BJIBAzMuMpgBAKABAaoBB2d3cy13aXrIAQjAAQE&amp;sclient=psy-ab&amp;ved=0ahUKEwiv7aKAmvfrAhVJTt8KHfPNBnUQ4dUDCAw&amp;uact=5</link>
    <dc:creator>Michael.Massing</dc:creator><dc:subject>polio scare postwar 50s 1950s 1956 epidemic outbreak risk public health</dc:subject>
<dc:source>https://pinboard.in/</dc:source>
<dc:identifier>https://pinboard.in/u:Michael.Massing/b:7138d5f5cd6f/</dc:identifier>
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	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:postwar"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:50s"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:1950s"/>
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	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:epidemic"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:outbreak"/>
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	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:public"/>
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</item>
<item rdf:about="https://www.medpagetoday.com/primarycare/dietnutrition/82492">
    <title>Is Everything We Know About Meat Consumption Wrong? | MedPage Today</title>
    <dc:date>2020-04-04T15:26:20+00:00</dc:date>
    <link>https://www.medpagetoday.com/primarycare/dietnutrition/82492</link>
    <dc:creator>Michael.Massing</dc:creator><dc:subject>meat diet risk processed evidence assessment meta-analysis systematic review</dc:subject>
<dc:source>https://pinboard.in/</dc:source>
<dc:identifier>https://pinboard.in/u:Michael.Massing/b:385fe8192571/</dc:identifier>
<taxo:topics><rdf:Bag>	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:meat"/>
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	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:risk"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:processed"/>
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	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:meta-analysis"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:systematic"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:review"/>
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</item>
<item rdf:about="https://www.medpagetoday.com/endocrinology/type1diabetes/82755?xid=nl_mpt_DHE_2019-10-16&amp;eun=g894600d0r">
    <title>Durable Glycemic Benefits With Islet Transplantation in T1D | MedPage Today</title>
    <dc:date>2020-04-04T15:18:28+00:00</dc:date>
    <link>https://www.medpagetoday.com/endocrinology/type1diabetes/82755?xid=nl_mpt_DHE_2019-10-16&amp;eun=g894600d0r</link>
    <dc:creator>Michael.Massing</dc:creator><description><![CDATA[Ten-year outcomes for 28 patients who underwent allogeneic islet transplantation with the Edmonton protocol showed 28% free from insulin and maintaining HbA1c levels of ≤6.5%, reported Marie-Christine Vantyghem, MD, PhD of the University of Lille in France, and colleagues.

Nearly 40% of these patients were able to achieve insulin independence with A1c ≤6.5% -- the study's primary endpoint -- only 5 years after islet transplantation, they wrote in Diabetes Care.]]></description>
<dc:subject>islet transplantation transplant pancreas T1D type 1 diabetes glycemic control blood glucose Edmonton protocol hypoglycemia unawareness kidney risk</dc:subject>
<dc:source>https://pinboard.in/</dc:source>
<dc:identifier>https://pinboard.in/u:Michael.Massing/b:29fec18ec123/</dc:identifier>
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	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:transplantation"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:transplant"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:pancreas"/>
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	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:control"/>
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	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:risk"/>
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</item>
<item rdf:about="https://jamanetwork.com/journals/jamainternalmedicine/article-abstract/2628970">
    <title>Heat-Not-Burn Tobacco Cigarettes: Smoke by Any Other Name | Tobacco and e-Cigarettes | JAMA Internal Medicine | JAMA Network</title>
    <dc:date>2020-02-05T02:34:38+00:00</dc:date>
    <link>https://jamanetwork.com/journals/jamainternalmedicine/article-abstract/2628970</link>
    <dc:creator>Michael.Massing</dc:creator><dc:subject>tobacco risk nicotine toxicology peer-reviewed research delivery system heated product heat-not-burn</dc:subject>
<dc:source>https://pinboard.in/</dc:source>
<dc:identifier>https://pinboard.in/u:Michael.Massing/b:47b29a56847a/</dc:identifier>
<taxo:topics><rdf:Bag>	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:tobacco"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:risk"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:nicotine"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:toxicology"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:peer-reviewed"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:research"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:delivery"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:system"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:heated"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:product"/>
	<rdf:li rdf:resource="https://pinboard.in/u:Michael.Massing/t:heat-not-burn"/>
</rdf:Bag></taxo:topics>
</item>
<item rdf:about="https://www.cdc.gov/tobacco/data_statistics/">
    <title>Data and Statistics | Smoking &amp; Tobacco Use | CDC</title>
    <dc:date>2020-02-05T02:32:42+00:00</dc:date>
    <link>https://www.cdc.gov/tobacco/data_statistics/</link>
    <dc:creator>Michael.Massing</dc:creator><dc:subject>secondhand smoke CDC risk data statistics smoking tobacco nicotine public health vaping</dc:subject>
<dc:source>https://pinboard.in/</dc:source>
<dc:identifier>https://pinboard.in/u:Michael.Massing/b:64a2e78c97c2/</dc:identifier>
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    <title>[untitled]</title>
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<item rdf:about="https://www.bloomberg.com/news/articles/2019-09-25/vaping-illness-signs-were-missed-or-ignored">
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    <title>A Vision for the Future - The Health Consequences of Involuntary Exposure to Tobacco Smoke - NCBI Bookshelf</title>
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    <title>Appendix - The Health Consequences of Involuntary Exposure to Tobacco Smoke - NCBI Bookshelf</title>
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<item rdf:about="https://www.ncbi.nlm.nih.gov/pubmed/20669524">
    <title>The Health Consequences of Involuntary Exposure to Tobacco Smoke: A Report of the Surgeon General - PubMed - NCBI</title>
    <dc:date>2020-02-05T01:35:53+00:00</dc:date>
    <link>https://www.ncbi.nlm.nih.gov/pubmed/20669524</link>
    <dc:creator>Michael.Massing</dc:creator><dc:subject>secondhand smoke Surgeon General HHS risk</dc:subject>
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<item rdf:about="https://www.who.int/tobacco/publications/prod_regulation/heated-tobacco-products/en/">
    <title>WHO | Heated tobacco products (HTPs) information sheet</title>
    <dc:date>2020-01-11T22:24:45+00:00</dc:date>
    <link>https://www.who.int/tobacco/publications/prod_regulation/heated-tobacco-products/en/</link>
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<item rdf:about="https://www.cdc.gov/tobacco/basic_information/e-cigarettes/pdfs/Electronic-Cigarettes-Infographic-508.pdf?s_cid=bb-osh-effects-graphic-007">
    <title>FDA. Electronic Cigarettes: What’s the Bottom Line?</title>
    <dc:date>2020-01-11T21:55:21+00:00</dc:date>
    <link>https://www.cdc.gov/tobacco/basic_information/e-cigarettes/pdfs/Electronic-Cigarettes-Infographic-508.pdf?s_cid=bb-osh-effects-graphic-007</link>
    <dc:creator>Michael.Massing</dc:creator><dc:subject>tobacco public health risk benefit e-cigarettes FDA</dc:subject>
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<item rdf:about="https://tobaccocontrol.bmj.com/content/tobaccocontrol/27/Suppl_1/s1.full.pdf">
    <title>Glantz SA inter alii. Tobacco Control 27 S1 Nov 2018 doi:10.1136/tobaccocontrol-2018-054601</title>
    <dc:date>2020-01-11T20:58:36+00:00</dc:date>
    <link>https://tobaccocontrol.bmj.com/content/tobaccocontrol/27/Suppl_1/s1.full.pdf</link>
    <dc:creator>Michael.Massing</dc:creator><dc:subject>HTP tobacco public health risk product FDA PMI IQOS international marketing addiction science criticism peer-reviewed research behavioral biological</dc:subject>
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<item rdf:about="https://www.who.int/tobacco/publications/prod_regulation/htps-marketing-monitoring/en/">
    <title>WHO | Heated tobacco products (HTPs) market monitoring information sheet</title>
    <dc:date>2020-01-11T20:39:12+00:00</dc:date>
    <link>https://www.who.int/tobacco/publications/prod_regulation/htps-marketing-monitoring/en/</link>
    <dc:creator>Michael.Massing</dc:creator><dc:subject>tobacco youth advertising prevalence use risk marketing WHO HTP</dc:subject>
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<item rdf:about="https://www.cdc.gov/tobacco/infographics/youth/index.htm">
    <title>Youth Tobacco Use Infographics | CDC</title>
    <dc:date>2020-01-11T20:33:23+00:00</dc:date>
    <link>https://www.cdc.gov/tobacco/infographics/youth/index.htm</link>
    <dc:creator>Michael.Massing</dc:creator><description><![CDATA[E-cigarette Use Among Youth is Rising as E-cigarette Advertising Grows]]></description>
<dc:subject>tobacco youth advertising e-cigarettes prevalence use risk</dc:subject>
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<item rdf:about="https://tobaccocontrol.bmj.com/content/27/Suppl_1#Researchpapers">
    <title>Table of contents | Tobacco Control</title>
    <dc:date>2020-01-11T19:15:11+00:00</dc:date>
    <link>https://tobaccocontrol.bmj.com/content/27/Suppl_1#Researchpapers</link>
    <dc:creator>Michael.Massing</dc:creator><description><![CDATA[November 2018 - Volume 27 - Suppl 1
Heated Tobacco Products]]></description>
<dc:subject>HTP tobacco public health risk product international marketing addiction science criticism peer-reviewed research</dc:subject>
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<dc:identifier>https://pinboard.in/u:Michael.Massing/b:052bcb2082c0/</dc:identifier>
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<item rdf:about="https://tobaccocontrol.bmj.com/content/27/Suppl_1/s7">
    <title>Heated tobacco products: things we do and do not know | Tobacco Control</title>
    <dc:date>2020-01-11T18:28:31+00:00</dc:date>
    <link>https://tobaccocontrol.bmj.com/content/27/Suppl_1/s7</link>
    <dc:creator>Michael.Massing</dc:creator><description><![CDATA[Most in the public health world would agree that the best evidence-based approaches should be applied to reduce death and illness due to tobacco use. Such approaches may include supporting addicted tobacco users to move to alternatives that are less harmful and truly reduce the population burden of tobacco diseases. It is important to bear in mind these two goals – alternative tobacco delivery devices may be less risky than combustible cigarettes for the individual smoker, but if they do not lead to a reduction in the prevalence of smoking there is no gain for public health.

What do we know about the safety of IQOS and the effect that introduction of these devices may have on smoking rates and the population burden of disease and premature mortality? Unfortunately information about IQOS and similar products is, at present, largely limited to industry reports. These include observations from marketing and data from product toxicity and human exposure studies. The papers in this Special Issue take a close look at the industry material, as well as the limited emerging academic literature on HTPs. The findings in broad terms are not terribly surprising. Data are scarce, and in particular, there are no long-term studies in human populations of the consequences of use of IQOS. Nevertheless an addictive product is being promoted by over-emphasising (or in some cases exaggerating) the limited evidence for its capacity to reduce harm, while minimising evidence on its potential toxicity. A sceptical view, conditioned by history, would be that this may be part of strategic efforts by the industry to retain existing consumers of tobacco products and generate new lifelong nicotine-dependent users. We offer some suggestions about what we do not know at present about IQOS, but need to understand to best inform tobacco control policies.]]></description>
<dc:subject>HTP tobacco public health risk regulation product development approval international marketing addiction</dc:subject>
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